Sunday, 3 March 2013

Currency vs. Rates Yo! | Wall Street Oasis

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Source: http://www.wallstreetoasis.com/forums/currency-vs-rates-yo

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Protection of the skin | Tammy's Health Articles

Advanced, affordable skin, all in an easy-to-carry stick protection!

Product review: this product is worth to learn about to consider the implementation as part of your plan health and well-being:

Camille, creator of Surf-Her skin care, launched a product revolutionary sun in March of this year. It is a ?unique and one of a type product? said Camille. ?I created this product in response to fellow surfers and athletes everywhere. We wanted to have the stronger more portable solar protection that can be used in water. ?It continues. His friends in the community surfing specifically requested a product to combat free radicals that cause premature aging and affections of the precancerous skin due to environmental stress. Enter, Surf ~ soil Nano Stick SPF 30!

Where do we need to reduce sun damage and minimize the risk of skin cancer more? Our all over the face, lips, ears and our eyes! ?The Nano stick is a 4 in 1 sunscreen! Camille shares, ?it offers a moisturizing formula that prevents drought and degradation of collagen. It is not irritating to the skin and can be used around the eyes. It offers high level of antioxidants to prevent radical damage free skin. And it also protects against UVA and UVB rays. The advanced formula is the most recent technology of nano with rich antioxidant rich chocolate, beta carotene and vitamin E to moisturize the skin while it protects. Camille explains, ?the formula is unique in the way that we mix a natural sunscreen with base and moisturizing antioxidants.?.

Users like the Nano Stick has a slight tint so it blends easily into any skin tone. Fans of Camille rave also that it can be applied around the eyes, without burning, and because it is small and portable, the stick can be easily slipped into the combination of diving or edge of short films for the extension of the application. Debra Trauntvein, a competitive surfer, is a big fan of Camille products and especially the Nano stick. It touts, ?it feels good and goes on the bar. Hard even after a 3 hour surf session!?Sencial skin

Camille was in the area of personal care for 30 years in the skin and the hair care industry. It is also been a surfer for more than 40 years, so who can better design the skin care and protection products as an expert who knows their needs directly the Sun! ?During the years my clients have asked me to develop products which, as well as protect the skin and hair from the harmful effects of the Sun.? Meet the needs of my clients and my friends, I created the Surf-her Skin Care & Surf ~ sunscreens soil. ?And now, we can all take advantage of its last contribution to the care of the skin and the world of sport, Surf ~ soil Nano Stick SPF 30!?

?

?

Source: http://www.tammyshealtharticles.com/protection-of-the-skin-in-a-stick/

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Saturday, 2 March 2013

Parkinson's disease: Parkin protects from neuronal cell death

Mar. 1, 2013 ? Researchers from Ludwig-Maximilians-Universitaet (LMU) in Munich identify a novel signal transduction pathway, which activates the parkin gene and prevents stress-induced neuronal cell death.

Parkinson's disease is the most common movement disorder and the second most common neurodegenerative disease after Alzheimer's disease. It is characterized by the loss of dopamin-producing neurons in the substantia nigra, a region in the midbrain, which is implicated in motor control. The typical clinical signs include resting tremor, muscle rigidity, slowness of movements, and impaired balance. In about 10% of cases Parkinson's disease is caused by mutations in specific genes, one of them is called parkin.

"Parkinson-associated genes are particularly interesting for researchers, since insights into the function and dysfunction of these genes allow conclusions on the pathomechanisms underlying Parkinson's disease," says Dr. Konstanze Winklhofer of the Adolf Butenandt Institute at the LMU Munich, who is also affiliated with the German Center for Neurodegenerative Diseases (DZNE). Winklhofer and her colleagues had previously observed that parkin can protect neurons from cell death under various stress conditions. In the course of this project, it became obvious that a loss of parkin function impairs the activity and integrity of mitochondria, which serve as the cellular power stations. In their latest publication, Winklhofer and coworkers uncovered the molecular mechanism that accounts for parkin's neuroprotective action.

"We discovered a novel signaling pathway that is responsible for the neuroprotective activity of parkin," Winklhofer reports. The central player of this pathway is a protein called NEMO, which is activated by the enzymatic attachment of a linear chain of ubiquitin molecules. This reaction is promoted by parkin, thereby enabling NEMO to activate a signal cascade, which ultimately leads to the expression of a specific set of genes. Winklhofer's team identified one essential gene targeted by this pathway, which turned out to code for the mitochondrial protein OPA1. OPA1 maintains the integrity of mitochondria and prevents stress-induced neuronal cell death.

"These findings suggest that strategies to activate this signal pathway or to enhance the synthesis of OPA1 in cells exposed to stress could be of therapeutic benefit," Winklhofer points out.

The newly identified signal pathway may also be relevant in the context of other neurological conditions that are characterized by the loss of specific neurons. Konstanze Winklhofer and her group are already engaged in further projects designed to determine whether other molecules regulated by this pathway might provide targets for therapeutic interventions.

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Story Source:

The above story is reprinted from materials provided by Ludwig-Maximilians-Universitaet Muenchen (LMU).

Note: Materials may be edited for content and length. For further information, please contact the source cited above.


Journal Reference:

  1. Anne?Kathrin M?ller-Rischart, Anna Pilsl, Patrick Beaudette, Maria Patra, Kamyar Hadian, Maria Funke, Regina Peis, Alexandra Deinlein, Carolin Schweimer, Peer-Hendrik Kuhn, Stefan?F. Lichtenthaler, Elisa Motori, Silvana Hrelia, Wolfgang Wurst, Dietrich Tr?mbach, Thomas Langer, Daniel Krappmann, Gunnar Dittmar, J?rg Tatzelt, Konstanze?F. Winklhofer. The E3 Ligase Parkin Maintains Mitochondrial Integrity by Increasing Linear Ubiquitination of NEMO. Molecular Cell, 2013; DOI: 10.1016/j.molcel.2013.01.036

Note: If no author is given, the source is cited instead.

Disclaimer: This article is not intended to provide medical advice, diagnosis or treatment. Views expressed here do not necessarily reflect those of ScienceDaily or its staff.

Source: http://feeds.sciencedaily.com/~r/sciencedaily/health_medicine/genes/~3/NkMYClxPbg4/130301122507.htm

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CMS and Healthcare Reform: What's the Connection? | HealthWorks ...

health reform and CMS TIGIThe Centers for Medicare and Medicaid Services (CMS) is a section of the United States Department of Health and Human Services (HHS). It is the largest administrator of health insurance benefits with its health care programs providing services to more than 100 million people, or a bit less than 1/3 of the United States Population. The programs administered by CMS include Medicare, Medicaid, and the Children's Health Insurance Program.

Historically, rules created by CMS for Medicare were often copied by private insurers. For instance, new tests for screening and/or diagnosing diseases were considered experimental until Medicare approved them for reimbursement. Most times private insurance companies would follow rather than lead in implementing payments for new diagnostics and medicines.

The role of The Centers for Medicare and Medicaid in implementing the Affordable Care Act (ACA) is quite important. They are the agency within HHS that is underwriting the reimbursement rules for implementing the ACA.

They are also the primary federal agency that is informing and educating the public about what to expect in the way of changes from implementation of the Affordable Care Act. Opponents to the ACA derisively call it "Obamacare," but the president and his supporters have said that is a nickname they are proud of.

Whether you support the ACA or not, it is the most significant change in health care since the implementation of Medicare and Medicaid fifty years ago.

The Centers for Medicare and Medicaid Services is concerned with three key components of the Affordable Care Act implantation. They are:

  1. Availability of health insurance
  2. Affordability of health insurance
  3. Reducing the cost of health care in the United States

They have developed programs to encourage all medical care facilities to computerize their medical record systems. The high dollar savings initially predicted have not yet materialized, although improved patient care due to electronic medical availability has been anecdotally reported.

It is no longer legal to bar someone from health insurance because they have a ?pre-existing? condition.

The lifetime caps (total amount of health insurance available to you) were done away with and no matter how expensive your care is you will receive it for as long as it is needed.

Children under age 26 living at home are also eligible for coverage.

CMS is tasked with making regulations to implement the ACA which is not yet popular with the majority of Americans. Even after the legislation passed, many are opposed to it. ?The law is being phased in over several years, with 2014 seen as the target date for full implementation.

While CMS is outstanding in its role of administrator for the plans of the Federal Government with very low administrative expenses it remains to be seen if they are up to the task of implementing the most sweeping reforms in health care insurance since Medicare.?

Image: freedigitalphotos.net

Authored by:

TIGI INC

Total Innovation Group, Inc. is a boutique consulting firm that provides performance improvement and business development consulting and training services. We help refine communications effectiveness and drive results.

See complete profile

Source: http://healthworkscollective.com/total-innovation-group-inc/86441/cms-and-healthcare-reform

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Disney Impressions ? Using Personal Tech in Walt Disney World ...

Personal technology has created a revolution in the way we work, play and communicate with each other when we are at work, home and on vacation. This is the first in a series of looks at what is available specifically in terms of personal technology usage in the parks. (Note for purposes of this series I will be dealing with Apple iOS products such as various flavors of iPhone and iPad. Please leave comments on application to other devices such as Android or Windows devices if you use one of them instead.)

I think that it is appropriate to spend some time discussing a philosophy of the use of personal tech while we are enjoying WDW. Let me state from the outset that I am a tech geek both by schooling and desire. I never met a technology that I didn?t love and have been an early adopter for many years including many variants that failed. (Beta video tapes, quadraphonic sound vinyl records, laserdisc players, Sony Minidiscs to name just a few. I plan to donate them all to the Smithsonian in my will?lol). Even so, I hate to see families where the younger kids are staring at Kid Icarus or Pokemon on their Nintendo 3DS while the teenagers are texting their BFFs all the while mom and dad are staring at the Park Map or Times Guide trying to figure out where to go or what show to see next, only to find the wait time is an hour when they get there.

What is wrong with that picture? Well, we have the kids totally disconnected from their families and the whole experience that is a Disney vacation. Next, we have frustrated parents who could use some help in planning their time in the Parks to have a much more enjoyable experience for the whole family. Fortunately, as the saying goes, there?s an app for that.

First let?s look at some non-Disney specific apps that may help to solve the disconnected kid problem. You can start by giving each kid a task that involves the use of some aspect of personal technology. For example instead of playing with the 3DS, give them the task of taking photos of the family, attractions etc. You can start with a simple digital camera and explain that they are creating a story of the family?s trip to The World. Later at home they can create a digital album on the computer or a printed book of the trip. Older siblings can assist with this and make it a project for all of the kids. They can also select some of the photos to create a calendar for the next year. Each month can explore aspects of the trip and help relive the memories for the future.

If your kids have a cell phone, they can take the photos with the phone and do as suggested above or use the photos in real time while in the Park. They could post individual photos on Facebook or create a Facebook photo album. One of them may want the responsibility of doing a check-in at each attraction either with Facebook or an app like foursquare or tagging people in photos. For a kid who has a creative bent, they can experiment with instagram and post photos they they have worked on. Similar opportunities are available with Twitter and google+. One note: if you do not like advertising that you are away from home for security reasons, be sure to check the security settings for the account where you are posting the information/photos so that only your friends can see them since presumably they know you are at Disney (and probably jealous as well).

If you are new to the Parks or just need a refresher on what is there, several of the more popular guides such as Birnbaum?s Official Guide, PassPorter?s Walt Disney World and The Unofficial Guide ? Walt Disney World are all available electronically either as e-books or downloadable pdf files. These can be placed on your iPhone, iPad or iPod Touch. I personally find them easier to read and use on a larger format such as an iPad Mini but any of the platforms can be made to work. The kids can use them to learn about the upcoming attraction they are in the queue for or look at the menu for a table service or counter serve restaurant for example. Parents can judge the suitability of an attraction for a very young child or get some idea of the pricing at a restaurant.

If you are a foodie and are interested in more information about what is available at a given restaurant and a review of it, I strongly recommend you subscribe to the Disney Food Blog. They have a wealth of info available on their website to give you a jump start on planning at home and their modestly priced downloadable e-books are a wealth of info you can carry on your portable device into the parks. They have both general WDW restaurant info as well as specialized books on the EPCOT Food and Wine Festival, EPCOT Snack Treats, and Holiday Treats among others.

In future posts I plan to cover both Disney and third party applications available for use in the Parks. I will attempt to give you unbiased advice on the use and usefulness (or lack thereof) of an app.

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Source: http://www.wdwfanzone.com/2013/03/disney-impressions-using-personal-tech-in-walt-disney-world-part-1/

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Friday, 1 March 2013

70% of parents want some form of integrated education> what's the ...

70% of parents want some form of integrated education> what?s the problem John? ? Slugger O'Toole

Conversation, politics and stray insights

Slugger is undergoing a facelift. Please have a look around and report any bugs you come across.

One of things Micheal Martin picked up on in his interview with Conor Bradford yesterday morning was the rather emphatic result of Lucid Talk?s poll in favour of integrated eduction:

In the latest Lucid Talk poll those in favour of children of all denominations sharing the same classroom stands at a massive 70%.

Tellingly, this figure represents almost identical proportions of Catholics and Protestants. On any other issue this would be accepted as the will of the people and politicians would act accordingly.

Martin asked, given the overwhelming majority of kids are still educated apart, what are blocks to making this happen? Indeed. Perhaps that?s a question that ought to be directed at the Education Minister, John O?Dowd?

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Copyright ? 2003 - 2013 Slugger O'Toole Ltd. All rights reserved.

Source: http://sluggerotoole.com/2013/03/01/70-of-parents-want-some-form-of-integrated-education-whats-the-problem-john/

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Wolf in sheep's clothing: Uncovering how deadly bacteria trick the immune system

Feb. 28, 2013 ? An outbreak of tuberculosis in the skid row area of downtown Los Angeles may have exposed up to 4,500 individuals to the bacterium that causes the deadly disease and has left federal officials scrambling to intervene.

The outbreak is occurring during winter, when homeless individuals are driven to crowded shelters, when influenza is peaking and when people's vitamin D levels, typically boosted by sunlight exposure, are low. A new UCLA study offers critical insight into how various bacteria may manipulate such factors to their advantage.

In a study published online Feb. 28 in the journal Science, UCLA researchers demonstrate that certain cunning bacteria -- including the type that causes tuberculosis -- can pretend to be viruses when infecting humans, allowing them to hijack the body's immune response so that they can hide out, unhindered, inside our cells. The findings may also help explain how viral infections like the flu make us more susceptible to subsequent bacterial infections such as pneumonia.

The study is particularly relevant to tuberculosis, which kills 1.4 million people worldwide each year. In the case of the recent Los Angeles outbreak, the findings could provide clues as to how the flu and a lack of vitamin D may have given the tuberculosis bacterium an edge.

"With 8.7 million in the world falling ill with tuberculosis each year, a better understanding of how these bacteria avoid our immune system could lead to new ways to fight them and to better, more targeted treatments," said senior author Dr. Robert L. Modlin, chief of dermatology at the David Geffen School of Medicine at UCLA and a professor of microbiology, immunology and molecular genetics in the UCLA Division of Life Sciences.

The protection our immune system provides against bacteria-based diseases and infections depends on the critical response of T cells -- white blood cells that play a central role in fighting infections -- and in particular on the release of a protein called interferon-gamma. Interferon-gamma utilizes the vitamin D hormone to alert and activate cells to destroy invading bacteria.

The research team found that bacteria can pretend to be viruses, triggering the immune system to launch an attack with a different protein, called interferon-beta, which is designed to fight viruses, not bacteria. Not only is interferon-beta ineffective against bacteria, but it can also block the action of interferon-gamma, to the advantage of bacteria. Further, if a real virus were to infect the body, triggering interferon-beta, it would divert the attention of the immune response, preventing an attack on the bacterial invader. The researchers say this may explain why the flu can lead to a more serious bacteria-based infection like pneumonia.

"Like a wolf in sheep's clothing, the bacteria can fool the immune system into launching an attack against the wrong type of infection, thus weakening the response against the bacteria," said first author Rosane M. B. Teles, a researcher in the dermatology division at the Geffen School of Medicine.

For the study, the team examined the mechanisms by which the virus-fighting interferon-beta protein suppresses the interferon-gamma defense response to bacterial infections, tricking the immune system into make the wrong defense choices.

The researchers studied leprosy as a model and then applied what they learned to understand tuberculosis, given that leprosy and tuberculosis are caused by related bacteria. Modlin noted that leprosy is an outstanding model for studying immune mechanisms in host defense since it presents as a clinical spectrum that correlates with the level and type of immune response of the pathogen.

The scientists first compared the genetic expression of the virus-fighting interferon-beta protein and the bacteria-fighting interferon-gamma protein in skin lesions from leprosy patients. They found that interferon-gamma was expressed in patients with the milder form of the disease and that interferon-beta was significantly increased in those with the more serious, progressive form of leprosy.

The researchers then compared the genes triggered by interferon-beta in these leprosy skin lesions with those found by two other groups of investigators in the blood of tuberculosis patients. Remarkably, there was a significant overlap. The interferon-beta genes were more frequent in both the skin lesions of leprosy patients with extensive disease and the blood of tuberculosis patients with more severe disease.

"We found this common interferon-beta gene pattern correlated with the greater extent of disease in both leprosy and tuberculosis, which are two very distinct diseases," Teles said.

Previous work by the UCLA team demonstrated that the interferon-gamma defense pathway relies on a specific mechanism involving vitamin D, a natural hormone that plays an essential role in the body's fight against infections. The current study found that interferon-beta suppressed elements involved in the interferon-gamma-triggered vitamin D pathway, preventing the immune system from killing the bacteria.

"The study raises the possibility that a decrease or increase of one of these two interferon proteins could shift the balance from mild to more serious disease," Modlin said. "We may find that therapeutic interventions to block or enhance specific interferon responses may be an effective strategy to alter the balance in favor of protection against bacterial diseases."

The new findings may indicate why, in winter, Los Angeles skid row residents are at an added disadvantage in dealing with tuberculosis -- for at least three reasons. First, because of colder weather at night, indigent homeless people tend to stay in shelters, where they live in close proximity with others, facilitating the spread of the infection. Second, due to the seasonal rise in influenza, the body's immune system could be diverted by the flu virus to produce interferon-beta, blocking an effective immune response to the tuberculosis bacteria. And finally, the drop in vitamin D levels associated with a decrease in exposure to sunlight during the winter months could diminish the ability of individuals' immune systems to kill the tuberculosis bacteria.

"With TB on the rise, this scenario could play out not only in cities in the United States but all over the world," Modlin said. "We hope that our findings may provide insight into harnessing new methods to combat TB and other bacterial infections as well."

Modlin noted that 8.7 million people become ill with tuberculosis each year and 1.4 million die from the disease. He said that an increase or decrease in one of the two interferon proteins could help explain why some people may be more resilient against, or susceptible to, the infection or have a more serious course of the disease.

The next step, according to Teles, is to further understand the mechanisms that bacterial pathogens use to activate interferon-beta and how bacteria can manipulate the immune system to block the potent interferon-gamma host antimicrobial responses in human infections.

The study was funded by the National Institute of Arthritis and Musculoskeletal Skin Diseases, part of the National Institutes of Health (NIH P50; ARO63020; RO1s AI022553, AR040312 and AI047868; and CTSA Grant UL1TR000124).

Additional authors are listed in the manuscript.

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Story Source:

The above story is reprinted from materials provided by University of California, Los Angeles (UCLA), Health Sciences. The original article was written by Rachel Champeau.

Note: Materials may be edited for content and length. For further information, please contact the source cited above.


Journal Reference:

  1. Rosane M. B. Teles, Thomas G. Graeber, Stephan R. Krutzik, Dennis Montoya, Mirjam Schenk, Delphine J. Lee, Evangelia Komisopoulou, Kindra Kelly-Scumpia, Rene Chun, Shankar S. Iyer, Euzenir N. Sarno, Thomas H. Rea, Martin Hewison, John S. Adams, Stephen J. Popper, David A. Relman, Steffen Stenger, Barry R. Bloom, Genhong Cheng, and Robert L. Modlin. Type I Interferon Suppresses Type II Interferon?Triggered Human Anti-Mycobacterial Responses. Science, 2013; DOI: 10.1126/science.1233665

Note: If no author is given, the source is cited instead.

Disclaimer: This article is not intended to provide medical advice, diagnosis or treatment. Views expressed here do not necessarily reflect those of ScienceDaily or its staff.

Source: http://feeds.sciencedaily.com/~r/sciencedaily/top_news/~3/J-rp2dbYRaM/130228155440.htm

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